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What are the symptoms of glomerulonephritis

2016-12-31 15:14
Nephritis is one of the common kidney diseases, nephritis is a serious renal disease. Bring great pain to patients, but also to the medical aspects of many thorny problems raised. So nephritis will also genetic? Experts pointed out: genetic nephritis is a monogenic genetic disease, the patient is heterozygous. There are different reports on the mode of inheritance. There are several kinds of gender reports: autosomal dominant inheritance. Incomplete sex linked dominant inheritance. Dominant segregation and chromosome association. The sex linked dominant inheritance.

Many patients know glomerulonephritis, but what are the symptoms of glomerulonephritis? There are a lot of patients are not very clear. Here are the main symptoms of glomerulonephritis:

1, lead to glomerular nephritis edema: glomerular filtration rate decreased, resulting in water and sodium retention; after protein resulted in decreased plasma colloid osmotic pressure loss, urine moisture retention; renin secretion, causing secondary aldosterone secretion, renal tubular sodium and water reabsorption factors increase, can cause renal edema. Light weight can only gain weight, body swelling, and abdominal cavity effusion (chest).

2, glomerulonephritis induced proteinuria: nephritis, increased glomerular capillary wall permeability, plasma albumin by membrane filtration, and proteinuria, so glomerulonephritis is the most important characteristic of proteinuria.

3, glomerulonephritis caused by tubular urine: tube type is formed in the urine protein coagulation and precipitation, so in nephritis, urine should have tube type. It should be noted that urine only protein and no tube type, can not reflect the glomerular lesions. Conversely, only tubular type without protein does not suggest glomerular lesions. Only in the urine both protein and tube type, before suggesting glomerular lesions.

4, glomerulonephritis induced hypertension: the kidney by regulating water, sodium metabolism, affecting the effective circulating blood volume, while generating renin, affecting the renin angiotensin aldosterone system and lead to high blood pressure. The incidence of hypertension in chronic nephritis was 33.3%, and the incidence of uremia was about 84%.

5, glomerulonephritis caused anemia: renal anemia often by water retention caused by dilution; glomerular severe lesions, red blood cell derived anemia and anemia. Anemia is generally normal form, normal pigment anemia.

6, glomerular nephritis caused by uremia and uremia: it is glomerular lesions to the end of the renal insufficiency, but not all patients with glomerulonephritis performance.
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