Vitamin E is widely present in foods and can be stored in various tissues in the body, so human vitamin E deficiency is relatively rare. It has been reported that vitamin E deficiency neurological symptoms do not occur until 5-10 years after human fat mal...
Vitamin E is widely present in foods and can be stored in various tissues in the body, so human vitamin E deficiency is relatively rare. It has been reported that vitamin E deficiency neurological symptoms do not occur until 5-10 years after human fat malabsorption.
Vitamins play an important role in growth and development. Vitamin e, together with carotenoids, vitamin c, selenium and glutathione, constitute a non-enzymatic antioxidant system in vivo.
Vitamin E, also known as tocopherol, is an essential nutrient for humans and higher animals and a fat-soluble vitamin. Representative of pure vitamin E: alpha-tocopherol, a yellow oily liquid, soluble in alcohol, fat and lipid solvents. It is stable to heat and acid, very sensitive to oxygen, and easily damaged when the grease is rancid. The general cooking method has little damage to vitamin E in food, but the activity of vitamin E significantly decreases when fried.
Vitamin E along with other antioxidants and antioxidant enzymes including superoxide dismutase, glutathione peroxidase, etc. together constitute an in vivo antioxidant system that protects biofilms and other proteins from free radical attack.
Many studies suggest that elevated oxidative status is closely related to the occurrence of some diseases, such as atherosclerosis, tumors, and aging. Animal studies and population studies suggest that vitamin E has the potential to prevent these oxidative damage-related diseases, but current population intervention studies still lack strong evidence.
The lipofuscin content in blood, vessel wall and cell tissue increased with age. Vitamin e supplementation could prevent lipid peroxidation from decreasing lipofuscin formation and delay senescence.
Vitamin E is an essential factor to maintain the integrity of red blood cells. Human deficiency of vitamin E reduces the number of red blood cells and shortens their survival time. The low level of vitamin E in the tissues of premature infants makes them prone to hemolytic anemia.Vitamin E is commonly used in clinical treatment.The human body does not need much vitamin E, but if it is lacking, it will also affect health.
Vitamin E deficiency is rare in humans, but it can occur in low-weight preterm infants, patients with blood β-lipoprotein deficiency, and fat malabsorption. In the absence of vitamin E, retinal degeneration, hemolytic anemia, muscle weakness, neurogenic degeneration, and cerebellar ataxia may occur.
If you have any problems on nephritic syndrome or kidney disease, you can consult with our online doctor or send email to us: email@example.com